Accumulation of b-1,3-glucanase and chitinase isoforms, and salicylic acid in the DL-b-amino-n-butyric acid-induced resistance response of pepper stems to Phytophthora capsici (Record no. 42249)

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control field MX-MdCICY
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control field 20250625124710.0
040 ## - CATALOGING SOURCE
Transcribing agency CICY
090 ## - LOCALLY ASSIGNED LC-TYPE CALL NUMBER (OCLC); LOCAL CALL NUMBER (RLIN)
Classification number (OCLC) (R) ; Classification number, CALL (RLIN) (NR) B-7937
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245 10 - TITLE STATEMENT
Title Accumulation of b-1,3-glucanase and chitinase isoforms, and salicylic acid in the DL-b-amino-n-butyric acid-induced resistance response of pepper stems to Phytophthora capsici
490 0# - SERIES STATEMENT
Volume/sequential designation Physiological and Molecular Plant Pathology, 51(5), p.305-322, 1997
520 3# - SUMMARY, ETC.
Summary, etc. Pepper (Capsicum annuum L.)plants sprayed with DL-b-amino-n-butyric acid (BABA)were protected against Phytophthora capsici infection. BABA treatment induced the synthesis and accumulation of b-1,3-glucanases and chitinases in the stem tissues of pepper plants. Their accumulation was very pronounced in the stems challenge-inoculated with P. capsici after BABA treatment. Several b-1,3-glucanase and chitinase isoforms accumulated in BABA treated P. capsici. When analysed by immunoblot of the denatured proteins, the 20 kDa b-1,3-glucanase and 32 kDa chitinase were found in pepper stems treated with BABA and}or infected by P. capsici. BABA treatment did not stimulate capsidiol production in pepper stems, but prior treatment led to high accumulation in P. capsici-infected ones. Unlike capsidiol production, BABA treatment triggered a dramatic increase in the endogenous levels of salicylic acid (SA)in pepper stems. The increase in endogenous SA was much pronounced in P. capsici infected stems after BABA treatment. In conclusion, the induction of resistance to P. capsici in pepper plants by BABA treatment positively correlated with the accumulation of certain b-1,3-glucanase and chitinase isoforms, and SA. These results suggest strongly that SA may act as an endogenous signal responsible for activating particular components of resistance to P. capsici and the induction of pathogenesis-related proteins such as b-1,3-glucanase and chitinase.
700 12 - ADDED ENTRY--PERSONAL NAME
Personal name Byung Kook Hwang
700 12 - ADDED ENTRY--PERSONAL NAME
Personal name Ji Yoon Sunwoo
700 12 - ADDED ENTRY--PERSONAL NAME
Personal name Young Jin Kim
700 12 - ADDED ENTRY--PERSONAL NAME
Personal name Beom Seok Kim
856 40 - ELECTRONIC LOCATION AND ACCESS
Uniform Resource Identifier <a href="https://drive.google.com/file/d/1HFCFlHcSEjtpqBXiYcdwatoCNuMUrRuZ/view?usp=drivesdk">https://drive.google.com/file/d/1HFCFlHcSEjtpqBXiYcdwatoCNuMUrRuZ/view?usp=drivesdk</a>
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  Clasificación local     Ref1 CICY CICY Documento préstamo interbibliotecario 25.06.2025   B-7937 25.06.2025 25.06.2025 Documentos solicitados