Capsaicin, a Component of Red Peppers, Inhibits the Growth of Androgen-Independent, p53 Mutant Prostate Cancer Cells (Record no. 47178)
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| fixed length control field | 02411nam a2200265Ia 4500 |
| 003 - CONTROL NUMBER IDENTIFIER | |
| control field | MX-MdCICY |
| 005 - DATE AND TIME OF LATEST TRANSACTION | |
| control field | 20250625153913.0 |
| 040 ## - CATALOGING SOURCE | |
| Transcribing agency | CICY |
| 090 ## - LOCALLY ASSIGNED LC-TYPE CALL NUMBER (OCLC); LOCAL CALL NUMBER (RLIN) | |
| Classification number (OCLC) (R) ; Classification number, CALL (RLIN) (NR) | B-12976 |
| 008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION | |
| fixed length control field | 250602s9999 xx |||||s2 |||| ||und|d |
| 245 10 - TITLE STATEMENT | |
| Title | Capsaicin, a Component of Red Peppers, Inhibits the Growth of Androgen-Independent, p53 Mutant Prostate Cancer Cells |
| 490 0# - SERIES STATEMENT | |
| Volume/sequential designation | Cancer Research, 66, p.3222-3229, 2006 |
| 520 3# - SUMMARY, ETC. | |
| Summary, etc. | Capsaicin is the major pungent ingredient in red peppers. Here, we report that it has a profound antiproliferative effect on prostate cancer cells, inducing the apoptosis of both androgen receptor (AR)-positive (LNCaP)and -negative (PC-3, DU-145)prostate cancer cell lines associated with an increase of p53, p21, and Bax. Capsaicin down-regulated the expression of not only prostate-specific antigen (PSA)but also AR. Promoter assays showed that capsaicin inhibited the ability of dihydrotestosterone to activate the PSA promoter/enhancer even in the presence of exogenous AR in LNCaP cells, suggesting that capsaicin inhibited the transcription of PSA not only via downregulation of expression of AR, but also by a direct inhibitory effect on PSA transcription. Capsaicin inhibited NF-K activation by preventing its nuclear migration. In further studies, capsaicin inhibited tumor necrosis factor-A-stimulated degradation of IKBA in PC-3 cells, which was associated with the inhibition of proteasome activity. Taken together, capsaicin inhibits proteasome activity which suppressed the degradation of IKBA, preventing the activation of NF-KB. Capsaicin, when given orally, significantly slowed the growth of PC-3 prostate cancer xenografts as measured by size [75 F 35 versus 336 F 123 mm3 (FSD); P = 0.017]and weight [203 F 41 versus 373 F 52 mg (FSD); P = 0.0006; capsaicin-treated versus vehicletreated mice, respectively]. In summary, our data suggests that capsaicin, or a related analogue, may have a role in the management of prostate cancer. |
| 700 12 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Mori, A. |
| 700 12 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Lehmann, S. |
| 700 12 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | O'Kelly, J. |
| 700 12 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Kumagai, T. |
| 700 12 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Desmond, J.C. |
| 700 12 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Pervan, M. |
| 700 12 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Mcbride, W.H. |
| 700 12 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Mcbride, W.H. |
| 700 12 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Koeffler, H.P. |
| 856 40 - ELECTRONIC LOCATION AND ACCESS | |
| Uniform Resource Identifier | <a href="https://drive.google.com/file/d/1DsDCV9gZCmvD4CUlFvM80sP2P_QD72FE/view?usp=drivesdk">https://drive.google.com/file/d/1DsDCV9gZCmvD4CUlFvM80sP2P_QD72FE/view?usp=drivesdk</a> |
| Public note | Para ver el documento ingresa a Google con tu cuenta: @cicy.edu.mx |
| 942 ## - ADDED ENTRY ELEMENTS (KOHA) | |
| Source of classification or shelving scheme | Clasificación local |
| Koha item type | Documentos solicitados |
| Lost status | Source of classification or shelving scheme | Damaged status | Not for loan | Collection | Home library | Current library | Shelving location | Date acquired | Total checkouts | Full call number | Date last seen | Price effective from | Koha item type |
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| Clasificación local | Ref1 | CICY | CICY | Documento préstamo interbibliotecario | 25.06.2025 | B-12976 | 25.06.2025 | 25.06.2025 | Documentos solicitados |
