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Differential gene expression profiles in the hippocampus of senescence-accelerated mouse

Tipo de material: TextoTextoSeries ; NeuroBiology of Aging, 28(4), p.497-506, 2007Trabajos contenidos:
  • Cheng, X
  • Zhou, W
  • Zhang, Y
  • Zhou, D
  • Yang, R
  • Chen, L
Tema(s): Recursos en línea: Resumen: The senescence-accelerated mouse (SAM)is an animal model for studying senescence and age-associated disorders due to its inherited aging phenotype. The SAM/prone8 (SAMP8)is a useful animal model to investigate the fundamental mechanisms involved in age-related learning and memory deficits that may have relevance to age-associated AD, while SAM/resistant1 (SAMR1)shows normal. To identify genes rendering the cognitive deterioration with aging, the subtractive cDNA libraries containing 1924 clones with the positive ratio of 96.18
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Documentos solicitados Documentos solicitados CICY Documento préstamo interbibliotecario Ref1 B-8947 (Browse shelf(Opens below)) Available

The senescence-accelerated mouse (SAM)is an animal model for studying senescence and age-associated disorders due to its inherited aging phenotype. The SAM/prone8 (SAMP8)is a useful animal model to investigate the fundamental mechanisms involved in age-related learning and memory deficits that may have relevance to age-associated AD, while SAM/resistant1 (SAMR1)shows normal. To identify genes rendering the cognitive deterioration with aging, the subtractive cDNA libraries containing 1924 clones with the positive ratio of 96.18

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