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245 1 0 _aInteractions of N-ethylmaleimide and aluminium fluoride with GABA B receptor function in rat neocortical slices
490 0 _vEuropean Journal of Pharmacology, 287(2), p.197-200, 1995
520 3 _aInteractions of N-ethylmaleimide and aluminium fluoride (AlF 4)with GABA B receptors have been examined using spontaneously discharging rat neocortical slices. The suppression of discharges by the GABA B receptor agonist baclofen (5-10 /zM)was irreversibly prevented by N-ethylmaleimide (10-50 tzM)and its analog N-phenylmaleimide (10-50 /zM), whilst superfusion of slices with NaF (10 mM)and A1C13 (100 ~M)to form a fluoroaluminate (AIF4-)complex markedly potentiated the action of baclofen. The lipoxygenase inhibitors, nordihydroguaiaretic acid (10-50/~M)and eicosatetraynoic acid (10-50/~M)or the phospholipase A 2 inhibitor bromophenacylbromide (50-100/~M)did not affect the response to baclofen. The depressant action of baclofen is evidently mediated through G-proteins, but is not dependent on arachidonic acid metabolites
650 1 4 _aGABA B RECEPTOR
650 1 4 _aBACLOFEN
650 1 4 _aNEOCORTICAL SLICE
650 1 4 _aRAT
650 1 4 _aN-ETHYLMALEIMIDE
650 1 4 _aALUMINUM FLUORIDE
650 1 4 _aG-PROTEIN
700 1 2 _aOng, J.
700 1 2 _aKerr, D.I.B.
856 4 0 _uhttps://drive.google.com/file/d/1OHnF-X51Igr236EQ13RHCiKkd0vTexay/view?usp=drivesdk
_zPara ver el documento ingresa a Google con tu cuenta: @cicy.edu.mx
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