000 02212nam a2200289Ia 4500
003 MX-MdCICY
005 20250625162418.0
040 _cCICY
090 _aB-18733
245 1 0 _aMedicinal species as MTDLs: Turnera diffusaWilld. Ex Schult inhibits CNS-enzymes and delays glutamate excitotoxicity in SH-SY5Y cells viaoxidative damage
490 0 _vFood and Chemical Toxicology, 106, p.466-476, 2017
520 3 _aTne of the most promising approaches to confront the complexity of central nervous system disorders are new multi-target directed ligands (MTDLs). Five medicinal species (Cereus grandiflorus (L.)Mill., Hyssopus officinalis L., Acorus calamus L., Silybum marianum L. Gaertn. and Turnera diffusa Willd. Ex Schult), selected for their ethnopharmacological relevance, were object for in vitroscreening. The aqueous extract of T. diffusa revealed the strongest neuroactive potential, inhibiting monoamine oxidase-A (IC 50 = 129.80 ± 11.97 µg/mL), and acetyl- and butyrylcholinesterase (IC 25 = 0.352 ± 0.011 and 0.370 ± 0.036 mg/mL, respectively). Its phenolic profile was established for the first time by HPLC-DAD-ESI/MSn. Twenty-six out of thirty-seven compounds were newly identified in this species. The pre-treatment with this flavonoidrich extract promoted a rightward shift of the glutamate concentration-neuronal cell (SH-SY5Y)death response curve. Furthermore, it significantly reduced the early phase formation of intracellular reactive species after glutamate and t-BHP exposure, suggesting that neuroprotection in SH-SY5Y cells was, in part, mediated by antioxidant mechanisms.
650 1 4 _aMEDICINAL SPECIES
650 1 4 _aTURNERA DIFFUSA
650 1 4 _aFLAVONOIDS
650 1 4 _aNEUROPROTECTION
650 1 4 _aCNS ENZYMES
650 1 4 _aGLUTAMATE EXCITOTOXICITY
700 1 2 _aBernardo, João
700 1 2 _aFerreres, Federico
700 1 2 _aGil-Izquierdo, Ángel
700 1 2 _aValentão, Patrícia
700 1 2 _aAndrade, Paula B.
856 4 0 _uhttps://drive.google.com/file/d/1AmK11mr07ZvBThs70IZPuaItaYqJ21Hz/view?usp=drivesdk
_zPara ver el documento ingresa a Google con tu cuenta: @cicy.edu.mx
942 _2Loc
_cREF1
008 250602s9999 xx |||||s2 |||| ||und|d
999 _c52879
_d52879