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090 _aB-19703
245 1 0 _aWAV E3 ubiquitin ligases mediate degradation of IAA32/34 in the TMK1-mediated auxin signaling pathway during apical hook development
490 0 _aProc. Natl. Acad. Sci., 121, p.e2314353121, 2024
500 _aArtículo
520 3 _aAuxin regulates plant growth and development through downstream signaling pathways, including the best-known SCFTIR1/AFB-Aux/IAA-ARF pathway and several other less characterized "noncanonical" pathways. Recently, one SCFTIR1/AFB-independent noncanonical pathway, mediated by Transmembrane Kinase 1 (TMK1), was discovered through the analyses of its functions in Arabidopsis apical hook development. Asymmetric accumulation of auxin on the concave side of the apical hook triggers DAR1-catalyzed release of the C-terminal of TMK1, which migrates into the nucleus, where it phosphorylates and stabilizes IAA32/34 to inhibit cell elongation, which is essential for full apical hook formation. However, the molecular factors mediating IAA32/34 degradation have not been identified. Here, we show that proteins in the CYTOKININ INDUCED ROOT WAVING 1 (CKRW1)/WAVY GROWTH 3 (WAV3) subfamily act as E3 ubiquitin ligases to target IAA32/34 for ubiquitination and degradation, which is inhibited by TMK1c-mediated phosphorylation. This antagonistic interaction between TMK1c and CKRW1/WAV3 subfamily E3 ubiquitin ligases regulates IAA32/34 levels to control differential cell elongation along opposite sides of the apical hook.
650 1 4 _aWAVY GROWTH 3
650 1 4 _aRING-FINGER E3 UBIQUITIN LIGASE
650 1 4 _aAUX/IAA PROTEINS
650 1 4 _aTRANSMEMBRANE KINASE 1
650 1 4 _a NONCANONICAL AUXIN SIGNALING
700 1 2 _aWang, J.-L.
700 1 2 _aWang, M.
700 1 2 _aZhang, L.
700 1 2 _aLi, Y.-X.
700 1 2 _aLi, J.-J.
700 1 2 _aLi, Y.-Y.
700 1 2 _aPu, Z.-X.
700 1 2 _aLi, D.-Y.
700 1 2 _aLiu, X.-N.
700 1 2 _aGuo, W.
856 4 0 _uhttps://drive.google.com/file/d/1VF3upb-Q5WmRlXzpBQ71ybXzY9XX_-9I/view?usp=drive_link
_zPara ver el documento ingresa a Google con tu cuenta: @cicy.edu.mx
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