| 000 | 03489nam a22004575i 4500 | ||
|---|---|---|---|
| 001 | 978-0-387-26283-3 | ||
| 003 | DE-He213 | ||
| 005 | 20250710083936.0 | ||
| 007 | cr nn 008mamaa | ||
| 008 | 100301s2006 xxu| s |||| 0|eng d | ||
| 020 |
_a9780387262833 _a99780387262833 |
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| 024 | 7 |
_a10.1007/b137303 _2doi |
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| 082 | 0 | 4 |
_a616.994 _223 |
| 100 | 1 |
_aDalgleish, Angus G. _eeditor. |
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| 245 | 1 | 4 |
_aThe Link Between Inflammation and Cancer _h[recurso electrónico] : _bWounds that do not heal / _cedited by Angus G. Dalgleish, Burkhard Haefner. |
| 264 | 1 |
_aBoston, MA : _bSpringer US, _c2006. |
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| 300 |
_aXII, 254 p. _bonline resource. |
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| 336 |
_atext _btxt _2rdacontent |
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| 337 |
_acomputer _bc _2rdamedia |
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| 338 |
_arecurso en línea _bcr _2rdacarrier |
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| 347 |
_atext file _bPDF _2rda |
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| 490 | 1 |
_aCancer Treatment and Research, _x0927-3042 ; _v130 |
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| 505 | 0 | _aForeword -- Inflammation and Cancer: The role of the immune response and angiogenesis -- Chronic Inflammation and Pathogenesis of GI and Pancreatic Cancers -- Cytokines, NF-kB, Microenvironment, Intestinal Inflammation and Cancer -- Regulation of NF-kB Transcriptional Activity -- The Role of Immune Cells in the Tumor Microenvironment -- Tumor-Microenvironment Interactions: The Selectin-Selectin Ligand Axis in Tumor-Endothelium Cross Talk -- CD95L/FasL and Trail in Tumour Surveillance and Cancer Therapy -- Infection & Neoplastic Growth 101: The required reading for microbial pathogens aspiring to cause cancer -- Cytokines as Mediators and Targets for Cancer Cachexia -- Targeting NF-kB in Anticancer Adjunctive Chemotherapy -- Index. | |
| 520 | _aThe transcription factor NF-kB has long been known to play a central role in the immune system by regulating the expression of key genes. Moreover, activation of this transcription factor helps a wide variety of cell types survive damage induced by pro-apoptotic stimuli. Owing to its critical role in the regulation of pro-inflammatory genes, NF-kB has long been regarded as a promising target for the discovery of anti-inflammatory drugs. More recently, NF-kB has emerged as a major culprit in a variety of human cancers mainly due to its ability to protect transformed cells from apoptosis. This finding should not have come as a surprise since there exists a link between inflammation and many types of cancer which was already suggested by Galen and later demonstrated by Virchow. This link, of crucial importance for the design of novel strategies for cancer treatment, is the topic of this book. Series Editor Cover Comments: "Inflammation is a known risk factor for a number of cancers. In this text, experts discuss the pathophysiology and molecular alterations that link these two processes. Potential therapeutics and preventive strategies are discussed." Steven T. Rosen, M.D. Series Editor | ||
| 650 | 0 | _aMEDICINE. | |
| 650 | 0 | _aONCOLOGY. | |
| 650 | 0 | _aMEDICAL LABORATORIES. | |
| 650 | 1 | 4 | _aMEDICINE & PUBLIC HEALTH. |
| 650 | 2 | 4 | _aONCOLOGY. |
| 650 | 2 | 4 | _aCANCER RESEARCH. |
| 650 | 2 | 4 | _aLABORATORY MEDICINE. |
| 700 | 1 |
_aHaefner, Burkhard. _eeditor. |
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| 710 | 2 | _aSpringerLink (Online service) | |
| 773 | 0 | _tSpringer eBooks | |
| 776 | 0 | 8 |
_iPrinted edition: _z9780387262826 |
| 830 | 0 |
_aCancer Treatment and Research, _x0927-3042 ; _v130 |
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| 856 | 4 | 0 |
_uhttp://dx.doi.org/10.1007/b137303 _zVer el texto completo en las instalaciones del CICY |
| 912 | _aZDB-2-SME | ||
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_2ddc _cER |
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_c56554 _d56554 |
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